Symptoms of facial paralysis may include: Unbalanced or asymmetric facial structures Inability to raise an eyebrow Inability to close an eyelid and protect the eyeball from drying out Looseness or sagging of the lower eyelid, leading to tears dripping down the cheek Inability to raise, lower or pucker the lips, leading to dribbling when drinking and slurring of some words when speaking The diagnosis of facial paralysis is often complicated. Facial paralysis may result from a disruption in the part of the brain called the motor cortex, injury to the facial nerve or damage to the muscles that control facial expression. Diagnosis involves a consultation with a doctor, a complete physical exam and imaging studies of the brain and face. Your doctor may recommend one or more of the following imaging tests: Magnetic resonance imaging MRI and computed tomography CT scan to rule out brain tumors, strokes and infections Electromyography EMG to evaluate the nerve and muscle Show More Treatment of facial paralysis depends on many factors, including the patient's age, cause of the paralysis, severity of paralysis and duration of symptoms.
Print Overview Bell's palsy causes sudden, temporary weakness in your facial muscles. This makes half of your face appear to droop. Your smile is one-sided, and your eye on that side resists closing. Bell's palsy, also known as facial palsy, can occur at any age. The exact cause is unknown.
Since it is sometimes hard to distinguish between a stroke and other causes of facial paralysis, it is a good idea to get your loved one to a doctor quickly if you notice facial paralysis. If you believe that you or a loved one may be experiencing a stroke, call as soon as possible. How is the cause of facial paralysis diagnosed? Be sure to discuss all your symptoms with your doctor, and share information about any other conditions or illnesses you may have.
Request new password Muscle Weakness Or Paralysis With Compromise Of Peripheral Nerve Muscle weakness or complete paralysis may be secondary to an interruption in the communication between the brain and nerve cells in the spinal cord that govern muscle activation, or to an interruption in the lower motor neuron or peripheral nerve [including the motor nerve cells in the spinal cord and their processes which connect with the muscle fibers], or both. If the motor axon, or nerve fiber, is inactivated by injection of a toxin that prevents transmission at the neuromuscular junction, the muscle will respond in the same fashion as if the motor nerve has degenerated. This same phenomenon may be observed when there is severe pathology of myelin, the insulating layer found around large sensory and motor nerve fibers.